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Search results (21)

« Back to Publications

Microglial inflammation and phagocytosis in Alzheimer's disease: Potential therapeutic targets

Nizami S. et al, (2019), British Journal of Pharmacology, 176, 3515 - 3532

A novel high-content imaging-based technique for measuring binding of Dickkopf-1 to low-density lipoprotein receptor-related protein 6

Priestley RS. et al, (2019), Journal of Pharmacological and Toxicological Methods, 95, 47 - 55

Targeting SHIP1 for therapeutic intervention in Alzheimer's disease

Mead E. et al, (2019), GLIA, 67, E587 - E588

Development of a high-content imaging-based technique to measure Dickkopf-1 binding to low-density lipoprotein receptor-related protein 6

Priestley RS. et al, (2019), BRITISH JOURNAL OF PHARMACOLOGY, 176, 2990 - 2990

Oscillating Magnet Array−Based Nanomagnetic Gene Transfection: A Valuable Tool for Molecular Neurobiology Studies

Subramanian M. et al, (2017), Nanomaterials, 7, 28 - 28

bnormalities in α/β-CaMKII and related mechanisms suggest synaptic dysfunction in hippocampus of LPA1 receptor knockout mice

Musazzi L. et al, (2011), The International Journal of Neuropsychopharmacology, 14, 941 - 953

Evaluation of expression and function of the H+/myo-inositol transporter HMIT

Di Daniel E. et al, (2009), BMC Cell Biology, 10

Neural network dysfunction in bipolar depression: clues from the efficacy of lamotrigine

Large CH. et al, (2009), Biochemical Society Transactions, 37, 1080 - 1084

Investigation of the H+–myo-inositol transporter (HMIT) as a neuronal regulator of phosphoinositide signalling

Daniel ED. et al, (2009), Biochemical Society Transactions, 37, 1139 - 1143

Prolyl oligopeptidase binds to GAP-43 and functions without its peptidase activity

Di Daniel E. et al, (2009), Molecular and Cellular Neuroscience, 41, 373 - 382

P.1.g.010 Regulation of neuronal growth cone morphology by prolyl oligopeptidase revealed by manipulation of gene expression

Di Daniel E. et al, (2007), European Neuropsychopharmacology, 17, S308 - S308

The common inositol-reversible effect of mood stabilizers on neurons does not involve GSK3 inhibition, myo-inositol-1-phosphate synthase or the sodium-dependent myo-inositol transporters

Di Daniel E. et al, (2006), Molecular and Cellular Neuroscience, 32, 27 - 36

nalysis of prolyl oligopeptidase (PO) as a target for bipolar disorder — characterisation of PO null-mutant mice

Di Daniel E. et al, (2006), European Neuropsychopharmacology, 16, S22 - S23

potential mechanism for the action of mood stabilizers involves prolyl oligopeptidase and its interaction with growth-associated protein

Di Daniel E. et al, (2006), BIPOLAR DISORDERS, 8, 61 - 61

Serotonin transporter gene polymorphism (5-HTTLPR) and emotional response to auditory hallucinations in schizophrenia

Sanjuan J. et al, (2005), The International Journal of Neuropsychopharmacology, 9, 131 - 131

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