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Search results (884)

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Characterisation of bacteria‐induced colitis and its modulation by probiotics in naked mole rats: a new mammalian model for acute inflammatory disease

Hart DW. et al, (2026), The Journal of Pathology

Promises under pressure: the modest predictive power of polygenic risk scores

Horton R. et al, (2026), European Journal of Human Genetics

Negative Selection Maintains Grossly Altered but Broadly Stable Karyotypes in Metastatic Colorectal Cancer.

Cross WCH. et al, (2026), Cancer discovery, OF1 - OF17

HLA Heterozygosity Influences Colorectal Cancer Risk and Survival Outcome.

Tsai Y-Y. et al, (2026), Gastroenterology

Mixed-model and transcriptome-wide association analyses identify transcription factors and genes associated with colorectal cancer susceptibility.

Chen Z. et al, (2026), Nature communications

Comparison between germline and somatic loss-of-function RNF43 mutations reveals different genotype-phenotype associations and provides insights into the genetic mechanisms of colorectal tumourigenesis.

Palles C. et al, (2025), Gut

Mathematical Modeling Quantifies "Just-Right" APC Inactivation for Colorectal Cancer Initiation.

Brunet Guasch M. et al, (2025), Cancer research, 85, 5113 - 5127

Loss of IDH1 and IDH2 mutations during the evolution of metastatic chondrosarcoma.

Cross W. et al, (2025), Genome biology, 26

Large-scale meta-analysis and precision functional assays identify FANCM regions in which PTVs confer different risks for ER-negative and triple-negative breast cancer.

Billaud A. et al, (2025), Breast (Edinburgh, Scotland), 85

Analysis of IDH1 and IDH2 mutations as causes of the hypermethylator phenotype in colorectal cancer

Ward JC. et al, (2025), The Journal of Pathology, 267, 40 - 55

Replication-associated mechanisms contribute to an increased CpG > TpG mutation burden in mismatch repair-deficient cancers

Ward JC. et al, (2025), Genome Medicine, 17

GWAS meta-analysis identifies five susceptibility loci for endometrial cancer.

Ramachandran D. et al, (2025), EBioMedicine, 118

Epithelial GREMLIN1 disrupts intestinal epithelial-mesenchymal crosstalk to induce a wnt-dependent ectopic stem cell niche through stromal remodelling

Mulholland EJ. et al, (2025), Nature Communications, 16

Supplementary Figure 2 from Coevolution of Atypical BRAF and KRAS Mutations in Colorectal Tumorigenesis

Woolley CE. et al, (2025)

Supplementary Table 5 from Coevolution of Atypical BRAF and KRAS Mutations in Colorectal Tumorigenesis

Woolley CE. et al, (2025)

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