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Publications

EGF and BMPs govern differentiation and patterning in human gastric glands

Wölffling S. et al, (2021), Gastroenterology

Molecular modelling of the gastric barrier response, from infection to carcinogenesis

Traulsen J. et al, (2021), Best Practice and Research: Clinical Gastroenterology

Genotoxic Effect of Salmonella Paratyphi A Infection on Human Primary Gallbladder Cells

Sepe LP. et al, (2020), mBio, 11

Helicobacter pylori-controlled c-Abl localization promotes cell migration and limits apoptosis.

Posselt G. et al, (2019), Cell Commun Signal, 17

The gastric "mucosoid cultures": a new model to investigate Helicobacter pylori infection and host response

Boccellato F. et al, (2018), HELICOBACTER, 23

Helicobacter pylori Depletes Cholesterol in Gastric Glands to Prevent Interferon Gamma Signaling and Escape the Inflammatory Response.

Morey P. et al, (2018), Gastroenterology, 154, 1391 - 1404.e9

Polarised epithelial monolayers of the gastric mucosa reveal insights into mucosal homeostasis and defence against infection.

Boccellato F. et al, (2018), Gut

Antiphospholipid antibodies detected by line immunoassay differentiate among patients with antiphospholipid syndrome, with infections and asymptomatic carriers.

Roggenbuck D. et al, (2016), Arthritis Res Ther, 18

Bacteria Moving into Focus of Human Cancer.

Boccellato F. and Meyer TF., (2015), Cell Host Microbe, 17, 728 - 730

ESTABLISHMENT OF AN IN VITRO INFECTION MODEL OF H-PYLORI USING HUMAN PRIMARY GASTRIC EPITHELIAL CELLS

Boccellato F. et al, (2014), HELICOBACTER, 19, 96 - 96

Differential regulation of miR-21 and miR-146a by Epstein-Barr virus-encoded EBNA2.

Rosato P. et al, (2012), Leukemia, 26, 2343 - 2352

Epstein-Barr virus encoded LMP1 downregulates TCL1 oncogene through miR-29b.

Anastasiadou E. et al, (2010), Oncogene, 29, 1316 - 1328

Epstein-Barr virus infection leads to partial phenotypic reversion of terminally differentiated malignant B cells.

Anastasiadou E. et al, (2009), Cancer Lett, 284, 165 - 174

EBNA2 interferes with the germinal center phenotype by downregulating BCL6 and TCL1 in non-Hodgkin's lymphoma cells.

Boccellato F. et al, (2007), J Virol, 81, 2274 - 2282

Epigenetic mechanisms do not control viral latency III in primary effusion lymphoma cells infected with a recombinant Epstein-Barr virus.

Anastasiadou E. et al, (2005), Leukemia, 19, 1854 - 1856

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