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Publications

A genome-wide association study of blood cell morphology identifies cellular proteins implicated in disease aetiology.

Akbari P. et al, (2023), Nature communications, 14

Author Correction: GWAS and meta-analysis identifies 49 genetic variants underlying critical COVID-19.

Pairo-Castineira E. et al, (2023), Nature, 619

Evaluation of T cell responses to naturally processed variant SARS-CoV-2 spike antigens in individuals following infection or vaccination.

Yin Z. et al, (2023), Cell reports, 42

GWAS and meta-analysis identifies 49 genetic variants underlying critical COVID-19.

Pairo-Castineira E. et al, (2023), Nature, 617, 764 - 768

Neutrophils and emergency granulopoiesis drive immune suppression and an extreme response endotype during sepsis.

Kwok AJ. et al, (2023), Nat Immunol

Comprehensive epigenomic profiling reveals the extent of disease-specific chromatin states and informs target discovery in ankylosing spondylitis

Brown AC. et al, (2023), Cell Genomics, 100306 - 100306

Persistence of inflammatory and vascular mediators 5 months after hospitalization with COVID-19 infection

Melhorn J. et al, (2023), Frontiers in Medicine, 10

Chromosome conformation capture approaches to investigate 3D genome architecture in Ankylosing Spondylitis

Davidson C. et al, (2023), Frontiers in Genetics, 14

Erratum: Transcriptomic Signatures in Sepsis and a Differential Response to Steroids. From the VANISH Randomized Trial (American Journal of Respiratory and Critical Care Medicine (2019) 199 (980–986) DOI: 10.1164/rccm.201807-1419OC)

Antcliffe DB. et al, (2022), American Journal of Respiratory and Critical Care Medicine, 206, 1572 - 1573

Association between age and the host response in critically ill patients with sepsis.

Michels EHA. et al, (2022), Critical care (London, England), 26

Natural Killer cells demonstrate distinct eQTL and transcriptome-wide disease associations, highlighting their role in autoimmunity

Gilchrist JJ. et al, (2022), Nature Communications, 13

COMBATdb: a database for the COVID-19 Multi-Omics Blood ATlas

Wang D. et al, (2022), Nucleic Acids Research

An immune dysfunction score for stratification of patients with acute infection based on whole-blood gene expression

Cano-Gamez E. et al, (2022), Science Translational Medicine, 14

Coronavirus disease 2019 subphenotypes and differential treatment response to convalescent plasma in critically ill adults: secondary analyses of a randomized clinical trial.

Fish M. et al, (2022), Intensive care medicine, 48, 1525 - 1538

Human leukocyte antigen alleles associate with COVID-19 vaccine immunogenicity and risk of breakthrough infection

Mentzer AJ. et al, (2022), Nature Medicine

Constructing custom-made radiotranscriptomic signatures of vascular inflammation from routine CT angiograms: a prospective outcomes validation study in COVID-19

Kotanidis CP. et al, (2022), The Lancet Digital Health, 4, e705 - e716

The suboptimal fibrinolytic response in COVID ‐19 is dictated by high PAI ‐1

Whyte CS. et al, (2022), Journal of Thrombosis and Haemostasis

Epigenomic analysis reveals a dynamic and context-specific macrophage enhancer landscape associated with innate immune activation and tolerance.

Zhang P. et al, (2022), Genome biology, 23

Redefining critical illness

Maslove DM. et al, (2022), Nature Medicine, 28, 1141 - 1148

SARS-CoV-2-specific antibody and T-cell responses 1 year after infection in people recovered from COVID-19: a longitudinal cohort study

Guo L. et al, (2022), The Lancet Microbe, 3, e348 - e356

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