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Publications

Hepcidin-Mediated Hypoferremia Disrupts Immune Responses to Vaccination and Infection

Frost JN. et al, (2021), Med, 2, 164 - 179.e12

Scoring a HAT-Trick against Lymphoma

Bannard O., (2019), Immunity, 51, 420 - 423

Germinal Center B Cells Replace Their Antigen Receptors in Dark Zones and Fail Light Zone Entry when Immunoglobulin Gene Mutations are Damaging.

Stewart I. et al, (2018), Immunity, 49, 477 - 489.e7

Germinal centers: programmed for affinity maturation and antibody diversification.

Bannard O. and Cyster JG., (2017), Curr Opin Immunol, 45, 21 - 30

Ubiquitin-mediated fluctuations in MHC class II facilitate efficient germinal center B cell responses.

Bannard O. et al, (2016), J Exp Med, 213, 993 - 1009

Phenotypic and Morphological Properties of Germinal Center Dark Zone Cxcl12-Expressing Reticular Cells.

Rodda LB. et al, (2015), J Immunol, 195, 4781 - 4791

Blockade of CTLA-4 promotes the development of effector CD8+ T lymphocytes and the therapeutic effect of vaccination with an attenuated protozoan expressing NY-ESO-1.

Dos Santos LI. et al, (2015), Cancer Immunol Immunother, 64, 311 - 323

Integrin-mediated interactions between B cells and follicular dendritic cells influence germinal center B cell fitness.

Wang X. et al, (2014), J Immunol, 192, 4601 - 4609

Germinal center centroblasts transition to a centrocyte phenotype according to a timed program and depend on the dark zone for effective selection.

Bannard O. et al, (2013), Immunity, 39, 912 - 924

MIR-17 similar to 92 promotes T follicular helper cell differentiation and represses subset-inappropriate gene expression

Baumjohann D. et al, (2013), CYTOKINE, 63, 246 - 247

The microRNA cluster miR-17∼92 promotes TFH cell differentiation and represses subset-inappropriate gene expression.

Baumjohann D. et al, (2013), Nat Immunol, 14, 840 - 848

CXCR4 promotes B cell egress from Peyer's patches.

Schmidt TH. et al, (2013), J Exp Med, 210, 1099 - 1107

Immunology. When less signaling is more.

Bannard OM. and Cyster JG., (2012), Science, 336, 1120 - 1121

Maintenance of T cell function in the face of chronic antigen stimulation and repeated reactivation for a latent virus infection.

Mackay LK. et al, (2012), J Immunol, 188, 2173 - 2178

Cutting edge: Virus-specific CD8+ T cell clones and the maintenance of replicative function during a persistent viral infection.

Bannard O. et al, (2010), J Immunol, 185, 7141 - 7145

Pathways of memory CD8+ T-cell development.

Bannard O. et al, (2009), Eur J Immunol, 39, 2083 - 2087

Secondary replicative function of CD8+ T cells that had developed an effector phenotype.

Bannard O. et al, (2009), Science, 323, 505 - 509

Fate-mapping identifies self-renewing effector CD8(+) T cells

Bannard O. et al, (2008), IMMUNOLOGY, 125, 64 - 64

A model for mapping the fate of CD8+T cells that have expressed granzyme B (GzmB)

Bannard OM. and Fearon D., (2008), FASEB JOURNAL, 22

Expression of the plasma cell transcriptional regulator Blimp-1 by dark zone germinal center B cells during periods of proliferation

BANNARD OLIVER. and RADTKE D., Frontiers in Immunology

Include forthcoming?