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Hypoxic and pharmacological activation of HIF inhibits SARS-CoV-2 infection of lung epithelial cells

Journal article

Wing PAC. et al, (2021), Cell Reports, 35, 109020 - 109020

Endothelial Oxygen Sensing in Alveolar Maintenance.

Journal article

Hodson E. and Ratcliffe PJ., (2020), American journal of respiratory and critical care medicine

Systemic silencing of Phd2 causes reversible immune regulatory dysfunction

Journal article

Yamamoto A. et al, (2019), Journal of Clinical Investigation, 129, 3640 - 3656

Conserved N-terminal cysteine dioxygenases transduce responses to hypoxia in animals and plants.

Journal article

Masson N. et al, (2019), Science, 365, 65 - 69

Nuclear entry and export of FIH are mediated by HIF1α and exportin1, respectively.

Journal article

Wang Y. et al, (2018), J Cell Sci, 131

Publisher Correction: The Jumonji-C oxygenase JMJD7 catalyzes (3S)-lysyl hydroxylation of TRAFAC GTPases

Journal article

Markolovic S. et al, (2018), Nature Chemical Biology, 14, 988 - 988

Studying the effects of disease associated polymorphism on a transcriptional pathway: A case study in renal cell carcinoma

Conference paper

Schmid V. et al, (2018), EUROPEAN JOURNAL OF HUMAN GENETICS, 26, 549 - 550

The Jumonji-C oxygenase JMJD7 catalyzes (3S)-lysyl hydroxylation of TRAFAC GTPases

Journal article

Markolovic S. et al, (2018), Nature Chemical Biology, 14, 688 - 695

New horizons in hypoxia signaling pathways in health and disease

Conference paper

Ratcliffe PJ., (2018), FREE RADICAL BIOLOGY AND MEDICINE, 120, S1 - S1

FACTOR INHIBITING HIF (FIH1) MODULATES CARDIAC FUNCTION AND METABOLISM Abstracts

Conference paper

Holliday J. et al, (2018), HEART, 104, A1 - A1

The SIN3A histone deacetylase complex is required for a complete transcriptional response to hypoxia

Journal article

Tiana M. et al, (2018), Nucleic Acids Research, 46, 120 - 133

Molecular and cellular mechanisms of HIF prolyl hydroxylase inhibitors in clinical trials.

Journal article

Yeh T-L. et al, (2017), Chem Sci, 8, 7651 - 7668

Prolyl hydroxylase 2 inactivation enhances glycogen storage and promotes excessive neutrophilic responses.

Journal article

Sadiku P. et al, (2017), J Clin Invest, 127, 3407 - 3420

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